Search results for "Smooth Muscle"

showing 10 items of 156 documents

Glucose-induced alterations of cytosolic free calcium in cultured rat tail artery vascular smooth muscle cells

1995

We have previously suggested that hyperglycemia per se may contribute to diabetic hypertensive and vascular disease by altering cellular ion content. To more directly investigate the potential role of glucose in this process, we measured cytosolic free calcium in primary cultures of vascular smooth muscle cells isolated from Sprague-Dawley rat tail artery before and after incubation with 5 (basal), 10, 15, and 20 mM glucose. Glucose significantly elevated cytosolic free calcium in a dose- and time-dependent manner, from 110.0 +/- 5.4 to 124.5 +/- 9.0, 192.7 +/- 20.4, and 228.4 +/- 21.9 nM at 5, 10, 15, and 20 mM glucose concentrations, respectively. This glucose-induced cytosolic free calci…

MaleTailmedicine.medical_specialtyVascular smooth muscleTime Factorschemistry.chemical_elementCalciumBiologyMuscle Smooth VascularImpaired glucose toleranceRats Sprague-DawleyCytosolLanthanumInternal medicinemedicineExtracellularAnimalsMannitolCells CulturedCellular calcium ion homeostasisDose-Response Relationship DrugGeneral MedicineArteriesmedicine.diseaseRatsCalcium ATPaseCytosolKineticsEndocrinologyGlucosechemistryCalciumMannitolmedicine.drugResearch Article
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Clinical implications of airway hyper-responsiveness in COPD

2006

COPD represents one of the leading causes of mortality in the general population. This study aimed at evaluating the relationship between airway hyperresponsiveness (AHR) and COPD and its relevance for clinical practice. We performed a MEDLINE search that yielded a total of 1919 articles. Eligible studies were defined as articles that addressed specific aspects of AHR in COPD, such as prevalence, pathogenesis, or prognosis. AHR appears to be present in at least one out of two individuals with COPD. The occurrence of AHR in COPD is influenced by multiple mechanisms, among which impairment of factors that oppose airway narrowing plays an important role. The main determinants of AHR are reduct…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtymedicine.medical_treatmentMEDLINEPopulationReviewsSettore MED/10 - Malattie Dell'Apparato RespiratorioBioinformaticsBronchial Provocation TestsPathogenesisDiagnosis DifferentialMedical Subject HeadingsPulmonary Disease Chronic ObstructivePredictive Value of TestsmedicineRespiratory HypersensitivityCOPDHumansairway smooth muscle aging bronchial obstructioneducationLungAsthmaCOPDeducation.field_of_studyLungbusiness.industryHealth Policyairway hyperresponsivenessSmokingPublic Health Environmental and Occupational HealthGeneral Medicinerespiratory systemmedicine.diseasePrognosisAsthmarespiratory tract diseasesbronchial hyperreactivitymedicine.anatomical_structurePredictive value of testsSmoking cessationbusinessAirwayInternational Journal of Chronic Obstructive Pulmonary Disease
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Therapeutic targets for overactive bladder other than smooth muscle

2015

For a long time, our concepts of regulation of urinary bladder function in health and disease as well as of the target structures of therapeutics have focused on detrusor smooth muscle cells. However, other structures including urothelium, afferent nerves and bladder blood vessels may also be important in pathophysiology and its treatment.Based on a selective review of literature, we discuss the role of urothelium, afferent nerve fibers and bladder blood vessels in bladder pathophysiology and as targets for treatment.There is solid evidence now that multiple anatomical structures within the urinary bladder contribute to the regulation of its function and hence may be targets for established…

Pathologymedicine.medical_specialtyMyocytes Smooth MuscleUrinary BladderClinical BiochemistryAnatomical structuresDiseaseurologic and male genital diseasesNerve FibersSmooth muscleAfferentDrug DiscoverymedicineAnimalsHumansMolecular Targeted TherapyUrotheliumPharmacologyAfferent PathwaysUrinary bladderUrinary Bladder Overactivebusiness.industrymedicine.diseasefemale genital diseases and pregnancy complicationsPathophysiologymedicine.anatomical_structureOveractive bladderDrug DesignMolecular MedicineUrotheliumbusinessNeuroscienceExpert Opinion on Therapeutic Targets
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PHOSPHODIESTERASE INHIBITORS PIROXIMONE AND ENOXIMONE INHIBIT PLATELET AGGREGATION IN VIVO AND IN VITRO

1997

The phosphodiesterase type III inhibitors piroximone (PIR) and enoximone (ENO) exert positive inotropic and vasodilating effects in patients with severe heart failure. PIR and ENO raise cyclic AMP levels in cardiac and vascular smooth muscle cells. Platelet activity is also regulated by intracellular levels of cyclic AMP. In this study we have investigated the effects of PIR and ENO on platelet activity in vivo and in vitro. PIR and ENO inhibited ADP induced platelet aggregation in a time- and concentration-dependent manner with IC50-values of 67 +/- 14 mumol/l and 129 +/- 6 mumol/l, respectively. Coincubation of PIR with the adenylate cyclase activator iloprost resulted in a synergistic po…

Blood PlateletsMalemedicine.medical_specialtyCardiotonic AgentsVascular smooth musclePlatelet AggregationPhosphodiesterase InhibitorsVasodilationIn vivoInternal medicineCyclic AMPmedicineAnimalsHumansEnoximonePlateletPlatelet activationRats WistarEnoximonebiologyChemistryImidazolesPhosphodiesteraseHematologyRatsEndocrinologyEnzyme inhibitorbiology.proteinCalciumPlatelet Aggregation Inhibitorsmedicine.drugThrombosis Research
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Tubulin-folding cofactor E deficiency is associated with vascular dysfunction and endoplasmatic reticulum stress of vascular smooth muscle cells

2021

Abstract Introduction Endothelial function assessed via flow mediated dilatation (FMD) has shown to predict risk in individuals with established cardiovascular diseases, whereas its predictive value is uncertain in the setting primary prevention. Purpose The aim of the current work was to discover and evaluate novel mediators of vascular dysfunction in the general population and in conditional knock-out transgenic animal models. Methods In order to identify novel targets that were negatively correlated with FMD and investigate their contribution in vascular function, a Genome Wide Association Study (GWAS) of 5,000 participants was performed and subsequently immune cell-, endothelial- and va…

Tubulin foldingVascular smooth musclebusiness.industryMedicineCOFACTOR ECardiology and Cardiovascular MedicinebusinessReticulumCell biologyEuropean Heart Journal
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Effects of sildenafil on human penile blood vessels.

2000

Abstract Objectives. To investigate the effects of sildenafil on human penile blood vessels and evaluate the interaction of sildenafil with neurogenic-mediated responses. Sildenafil is currently used in the treatment of erectile dysfunction. Methods. Penile dorsal arteries and deep dorsal veins were obtained from 14 multiorgan donors. Vascular rings were suspended in organ bath chambers, and the isometric tension was recorded. We then studied the effects of sildenafil on precontracted vessels and the neurogenic (noradrenergic and nitrergic) responses. Results. Sildenafil (10 −9 to 3 × 10 −6 M) caused concentration-dependent relaxation and amplified the relaxation induced by sodium nitroprus…

AdultMalemedicine.medical_specialtyAdolescentSildenafilPhosphodiesterase InhibitorsUrologyMuscle RelaxationPiperazinesSildenafil CitrateNitric oxideVeinschemistry.chemical_compoundInternal medicineMedicineHumansSulfonesGuanethidinebusiness.industrySmooth muscle contractionArteriesMiddle AgedPDE5 drug designrespiratory tract diseasesVasodilationEndocrinologymedicine.anatomical_structurechemistryPurinesVasoconstrictioncardiovascular systemSodium nitroprussidebusinessZaprinastBlood vesselmedicine.drugMuscle ContractionPenisUrology
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A novel in vitro model for the study of plaque development in atherosclerosis

2006

SummaryFor the study of atherogenesis in vitro, coculture systems have been devised, in which two or more cell types can be cultured in close contact to each other. Herein, we describe a novel in vitro model that aims at the simulation of the morphology ofa normal muscular artery allowing for the study of the initial events in atherosclerosis. Usinga modified fibrin gel as a scaffold for the coculture of endothelial cells (ECs) and smooth muscle cells (SMCs), we generated an autologous in vitro model with a multilayer growth of SMCs (intima-like structure) covered by an endothelium. The production of extracellular matrix (ECM) could be visualized histologically and verified by (i) ascorbic-…

Pathologymedicine.medical_specialtyTime FactorsEndotheliumCellular differentiationMyocytes Smooth MuscleMonocytesMuscle Smooth VascularCell LineExtracellular matrixCell MovementLamininCell AdhesionmedicineHumansFoam cellFibrinDose-Response Relationship Drugbiologybusiness.industryEndothelial CellsCell DifferentiationHematologyAtherosclerosisCoculture TechniquesIn vitroExtracellular MatrixCell biologyLipoproteins LDLmedicine.anatomical_structureCell culturebiology.proteinbusinessGelsFoam CellsLipoproteinThrombosis and Haemostasis
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Type 5 phosphodiesterase (PDE5) and the vascular tree: from embryogenesis to aging and disease

2020

Highlights • Vascular development depends on the timely differentiation of endothelial and smooth muscle cells, that mutually influence their developmental fate. • Endothelial and vascular smooth muscle cell (VSMC) compartments can mutually influence cell and tissue modifications during vascular aging and in vascular disease. • Keeping in mind that PDE5 is mainly expressed in VSMCs, we surveyed the literature on the role of PDE5 in vascular development, aging and disease. • Although most results have been obtained by PDE5 pharmacological inhibition, no data are available, to date, on vascular development, aging or disease following PDE5 genetic ablation.

0301 basic medicineCell typeAgingVascular smooth muscleMyocytes Smooth MuscleVSMCsEmbryonic DevelopmentECsContext (language use)DiseaseBiologyMuscle Smooth VascularArticle03 medical and health sciences0302 clinical medicinenitric oxidevascular smooth muscle cellsHumansBioresorbable vascular scaffoldCyclic Nucleotide Phosphodiesterases Type 5ECEmbryogenesisPhosphodiesteraseVascular agingCell biologycGMPSettore MED/23ECs; PDE5; VSMCs; cGMP; nitric oxide030104 developmental biologyVascular aging; vascular smooth muscle cells; phosphodiesterasePDE5phosphodiesterase030217 neurology & neurosurgeryFunction (biology)Developmental Biology
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Potential Functional Significance of Brain-Type and Muscle-Type Nitric Oxide Synthase I Expressed in Adventitia and Media of Rat Aorta

1999

Abstract —Skeletal muscle and myocardium express μNOS I, an elongated splice variant of neuronal-type nitric oxide (NO) synthase (NOS I), and NOS III, endothelial-type NO synthase, respectively. This study was designed to elucidate whether vascular smooth muscle also contains a constitutively expressed NO synthase isoform. In the rat, μNOS I contains an insert of 102 nucleotides after nucleotide 2865 of the cDNA, yielding a protein of 164 kd. Reverse transcription-polymerase chain reaction with primers flanking this insert and with insert-specific primers indicated that endothelium-denuded rat aorta expresses both brain-type NOS I and μNOS I. RNase protection analyses with an antisense RNA…

Gene isoformPathologymedicine.medical_specialtyDNA ComplementaryVascular smooth muscleNitric Oxide Synthase Type IIIBlotting WesternAorta ThoracicNitric Oxide Synthase Type INitroarginineGene Expression Regulation EnzymologicMuscle Smooth VascularMembrane PotentialsPotassium ChlorideNitric oxideImmunoenzyme TechniquesRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundmedicine.arteryAdventitiamedicineAnimalsVasoconstrictor AgentsAorta AbdominalRNA MessengerMuscle SkeletalMessenger RNAAortabiologyBrainSkeletal muscleMolecular biologyRatsNitric oxide synthaseAntisense Elements (Genetics)medicine.anatomical_structurechemistryVasoconstrictionbiology.proteinCalciumFemaleNitric Oxide SynthaseTunica MediaCardiology and Cardiovascular MedicineArteriosclerosis, Thrombosis, and Vascular Biology
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Immunohistochemical characterization of endometriosis-associated smooth muscle cells in human peritoneal endometriotic lesions

2011

Background Smooth muscle cells (SMC) are common components of endometriotic lesions. SMC have been characterized previously in peritoneal, ovarian and deep infiltrating endometriotic lesions and adenomyosis. The aim of this retrospective study was to investigate the extent of differentiation in endometriosis-associated SMC (EMaSMC) in peritoneal endometriotic lesions. Methods We obtained biopsies from peritoneal endometriotic lesions (n = 60) and peritoneal sites distant from the endometriotic lesion (n = 60), as well as healthy peritoneum from patients without endometriosis (control tissue, n = 10). These controls were hysterectomy specimens from patients without endometriosis or adenomyos…

AdultendometriosisPathologymedicine.medical_specialtyBiopsyEndometriosisEstrogen receptorsmooth muscle metaplasiaPeritoneumRetrospective StudieProgesterone receptormedicinevasopressin receptorHumansMyocyteAdenomyosisEndometriosiOxytocin receptorRetrospective Studiesendometriosis; Oxytocin receptor; smooth muscle metaplasiaMyosin Heavy Chainsbusiness.industryRehabilitationMyosin Heavy ChainObstetrics and GynecologyCell DifferentiationMuscle SmoothMiddle Agedmusculoskeletal systemmedicine.diseaseSettore MED/40 - Ginecologia E OstetriciaImmunohistochemistrymedicine.anatomical_structureGene Expression RegulationPremenopauseReceptors EstrogenReproductive MedicineReceptors Oxytocinsmooth muscle actinImmunohistochemistryFemaleDesminPeritoneumReceptors ProgesteronebusinessHumanHuman Reproduction
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